BSE, CJD quotes
Quotes   Mark Purdey   BSE, CJD

See: Pesticides & disease quotes

[2009 April] Pandemic of panic - After salmonella, bird flu, the Millennium Bug... should we actually be scared this time?    In 1996, when the greatest food scare of all exploded over BSE, front-page headlines greeted the admission on Newsnight by the Government's chief scientist John Pattison that the death toll from CJD caught by eating beef could within a few years reach 500,000. A year later, however, scarcely any attention at all was paid to Dr Pattison's confession that he had now revised his figure downwards to just '100' (indeed, it now seems highly likely that beef was not the cause of CJD at all).

Research programs on Hepatitis C, BSE, SARS, Avian flu and current vaccination policies all developed along the same logic, that of maximizing financial profits. Whenever we try to understand how some highly questionable therapeutic policies have been recommended at the highest levels of public health authorities (WHO, CDC, RKI etc.), we frequently discover either embarrassing conflicts of interests, or the lack of essential control experiments, and always the strict rejection of any open debate with authoritative scientists presenting dissident views of the pathological processes. Manipulations of statistics, falsifications of clinical trials, dodging of drug toxicity tests have all been repeatedly documented. All have been swiftly covered up, and none have been able to, so far, disturb the cynical logic of today’s virus research business. [2008] Virus Mania Foreword by Etienne de Harven

But if a toxin can speed up the outbreak of a disease, like alcohol can contribute to liver diseases, then it can also be the sole cause. However, if Phosmet would be declared as cause of BSE, compensation lawsuits in billions would wait for both the British government and the manufacturer of the insecticide. This is certainly not desirable for them, so they prefer to surround the basically clear context in a fog of prions.
    Intoxication hypotheses are easily testable and in contrast to the virus or prion hypotheses also falsifiable. They can be examined toxicologically and epidemiologically and then we can either accept or reject them. BSE/AIDS/Hepatitis C Infectious or Intoxication Diseases? By Claus Köhnlein

"The levels of manganese are naturally low in the soils of the Quinaborough area but the point is that the surrounding farmers are spraying on a potent manganese fertiliser several times a year. There is also widespread application of sewage sludge around all the villages that have a variant CJD problem. Manganese is also at a high level in sewage and it becomes air borne during spreading operations. Villages will therefore be breathing in these various manganese substances and taking it straight into their brains via the nasal/olfactory route."--Mark Purdey

"Science has made a monumental blunder. Mad cow disease— bovine spongiform encephalopathy, or BSE—is not an infection spread by a pathogen, that is, a virus or a bacteria. It is a naturally occurring degenerative disease—a disease of old age— that is accelerated by toxic poisoning."----Dr Anthony and Benjamin Parish

"1952-53: Several bio-weapons were employed by the U.S. in Korea, including brucellosis. Evidence also suggests that a pathogen causing hemorrhagic fever was deployed along the Hantaan River, but it 'blew back' over American troops, killing several hundred. D. Carleton Gajdusek (see article by Ms. Heslin this issue) sent by Pentagon to help contain the damage.
1957: Carleton Gajdusek turned up in remote New Guinea highlands where hundreds of Fore tribe were suffering from Creutzfeldt-Jakob disease (kuru).."--Scott

"There are more explosive implications to his -and other's latest research.   Purdey says similar organophosphate-induced protein deformation could also underlie Alzheimer's and related diseases. If that were true, the litigation fallout would destroy some pharmaceutical giants, and a lot of very influential noses would be out of joint.  In 1996, leading Alzheimer's researcher Tsunao Saitoh, 46 and his 13 -year-old daughter were killed in La Jolla, California, in what a Reuters report described as a "very professionally done" shooting.  What Alzheimer's Disease, Mad Cow Disease, and CJD have in common, is abnormal brain proteins and a putative link to organophosphates."---Fintan Dunne

I said to myself: "This is a professional hit. This is not a random killing by muggers." (My judgement was later borne out by a Reuters report [5/11/96, San Diego] which called the double murder "very professionally done.") I was also becoming familiar with violence against scientists and others victims who were all involved in one way or another with abnormal brain proteins. Some of them implicated a chemical in Mad Cow Disease and its species-transmissible form, including the human version, CJD. Purdey's house was burned down and his lawyer who was working with him on Mad Cow Disease had been driven off the road by another vehicle and subsequently died. The veterinarian on the case also died in a car crash. Purdey's new lawyer, too, had a car accident, but not fatal. Dr. C. Bruton, a CJD specialist -- who had just produced a paper on the a new strain of CJD -- was killed in a car crash before his work was announced to the public. Purdey speculates that Bruton might have known more than what was revealed in his paper. PROFESSIONAL HIT ON SAITOH, WORLD CLASS SCIENTIST!  By Claire W. Gilbert, Ph.D

I'M APPALLED by the unfounded assumptions of 'experts' about the hyper-infectious nature of the BSE/CJD agent (Mail). Basic evidence suggests variant CJD didn't arise from eating BSE-affected cattle. Third World countries imported even greater quantities of the incriminated British meat than we consumed here, but no cases of vCJD have occurred in those countries. I've travelled worldwide carrying out analyses of areas with prion diseases, of which vCJD is one. These diseases increase only in areas with a high manganese/low copper imbalance. Experiments at Cambridge and Case Western, Cleveland, universities confirmed my findings that misshapen prions develop whenever brain cells are exposed to this imbalance.
I found one organophosphate — poured on cows to counteract warble fly and on humans for headlice — which bonded with copper in the brain, starving the prion protein of its copper partner. When this happens, foreign metals, such as manganese, are more likely to bond with prions, which can have devastating consequences. Manganese is increasing in the environment due to its use as a fertiliser and fungicide, a lead substitute in petrol, and emission from factories. It was fed to UK cattle in calf milk replacement powder (at levels up to 1,000 times higher than in normal cow's milk) and as supplements for bone growth. Manganese is also fed to humans (in health food supplements), deer (for antler growth) and cats —and all have had prion disease. The totalitarian mindset about the origins of prion diseases betrays an agenda. It's more about global profits for the GM soya protein empires than about the illusory health risks of animal proteins to humans.